{"schemaVersion":"peptidescanner-evidence-object/v1","objectId":"story:pubmed-pmid-39149351-glucagon:v1","objectPathId":"pubmed-pmid-39149351-glucagon-v1","kind":"story-version","urls":{"story":"https://peptidescanner.com/stories/pubmed-pmid-39149351-glucagon/","json":"https://peptidescanner.com/evidence/v1/objects/pubmed-pmid-39149351-glucagon-v1.json","markdown":"https://peptidescanner.com/evidence/v1/objects/pubmed-pmid-39149351-glucagon-v1.md"},"story":{"slug":"pubmed-pmid-39149351-glucagon","version":1,"domain":"Literature metadata","dateInSource":"2026-07-26","sourceCheckedAt":"2026-08-10","publishedAt":"2026-08-13","peptideSlugs":["glucagon"],"peptides":[{"slug":"glucagon","url":"https://peptidescanner.com/peptides/glucagon/"}]},"claims":[{"id":"headline","text":"Interruption of glucagon signaling augments islet non-alpha cell proliferation in SLC7A2- and mTOR-dependent manners.","sourceIds":["pubmed-observation-pmid-39149351-2026-07-26-463dfd06aa39"]},{"id":"summary","text":"PubMed lists PMID 39149351 in bioRxiv : the preprint server for biology as Journal Article, Preprint, with publication-date metadata of 2024-08-07 (day precision). PeptideScanner associated the retained record with the identity slug glucagon. These fields reproduce attributed bibliographic metadata and do not summarize or endorse the article's findings.","sourceIds":["pubmed-observation-pmid-39149351-2026-07-26-463dfd06aa39"]},{"id":"paragraph-1","text":"PubMed lists PMID 39149351 in bioRxiv : the preprint server for biology as Journal Article, Preprint, with publication-date metadata of 2024-08-07 (day precision). PeptideScanner associated the retained record with the identity slug glucagon. These fields reproduce attributed bibliographic metadata and do not summarize or endorse the article's findings.","sourceIds":["pubmed-observation-pmid-39149351-2026-07-26-463dfd06aa39"]},{"id":"paragraph-2","text":"This page reproduces bounded bibliographic metadata attributed to PubMed. Its title and publication details are source metadata, not PeptideScanner's endorsement or an assertion of safety, efficacy, or scientific truth.","sourceIds":["pubmed-observation-pmid-39149351-2026-07-26-463dfd06aa39"]}],"sources":[{"id":"pubmed-observation-pmid-39149351-2026-07-26-463dfd06aa39","name":"PubMed","sourceClass":"A","title":"Interruption of glucagon signaling augments islet non-alpha cell proliferation in SLC7A2- and mTOR-dependent manners. · retained source version 1:2026-07-26:current:463dfd06aa397cf3a5db6056cc0ac88af007abe639c3df814a3ac48f09aa3751","url":"https://pubmed.ncbi.nlm.nih.gov/39149351/"}],"correction":{"state":"none"},"boundaries":{"scope":"selected-public-evidence-only","reporting":"PeptideScanner reproduces bounded bibliographic metadata attributed to PubMed. It does not summarize or endorse the article's findings.","unknowns":["Sources outside the selected public PeptideScanner bundle may exist and are not represented by this object.","This object does not determine whether cited records agree, corroborate one another, or establish scientific truth.","This object does not determine safety, efficacy, causality, or suitability for any person."],"methodologyUrl":"https://peptidescanner.com/methodology/","medicalDisclaimerUrl":"https://peptidescanner.com/medical-disclaimer/"},"commitments":{"publicationLedgerSha256":"d8e3373c7ac19bb421e90b61d2430e4f786598d7c86839ab78a38bd7d2b0e533","tierARecordSha256":"92f72d3e04405f32143f3a01e3f57f37ae491f12dbb922f41f9a24e3544d33ee"},"integrity":{"algorithm":"sha256","contentSha256":"8f3d30bf7d9c3a28ea1f14dce4caffb1544da0c4f0f33391d58f12860678d634"}}
