# N-terminal degradation of ACTH(4-10) and its synthetic analog semax by the rat blood enzymes.

> PubMed lists PMID 1851003 in Biochemical and biophysical research communications as Journal Article, with publication-date metadata of 1991-04-30 (day precision). PeptideScanner associated the retained record with the identity slug semax. These fields reproduce attributed bibliographic metadata and do not summarize or endorse the article's findings.

- Evidence object: `story:pubmed-pmid-1851003-semax:v1`
- Evidence domain: Literature metadata
- Date in source: 2019-06-12
- Source checked: 2026-07-30
- Published by PeptideScanner: 2026-08-13
- Story landing page: https://peptidescanner.com/stories/pubmed-pmid-1851003-semax/
- JSON record: https://peptidescanner.com/evidence/v1/objects/pubmed-pmid-1851003-semax-v1.json
- Content SHA-256: `2f3694be65df34a5700e6601ccbd6cc6b40afb12f566d6c526984b57b932ee72`

## Claims and source pins

### Headline

N-terminal degradation of ACTH(4-10) and its synthetic analog semax by the rat blood enzymes. ([`pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8`](#source-pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8))

### Summary

PubMed lists PMID 1851003 in Biochemical and biophysical research communications as Journal Article, with publication-date metadata of 1991-04-30 (day precision). PeptideScanner associated the retained record with the identity slug semax. These fields reproduce attributed bibliographic metadata and do not summarize or endorse the article's findings. ([`pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8`](#source-pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8))

### Paragraph 1

PubMed lists PMID 1851003 in Biochemical and biophysical research communications as Journal Article, with publication-date metadata of 1991-04-30 (day precision). PeptideScanner associated the retained record with the identity slug semax. These fields reproduce attributed bibliographic metadata and do not summarize or endorse the article's findings. ([`pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8`](#source-pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8))

### Paragraph 2

This page reproduces bounded bibliographic metadata attributed to PubMed. Its title and publication details are source metadata, not PeptideScanner's endorsement or an assertion of safety, efficacy, or scientific truth. ([`pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8`](#source-pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8))

## Sources

<a id="source-pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8"></a>
- **N-terminal degradation of ACTH(4-10) and its synthetic analog semax by the rat blood enzymes. · retained source version 1:2019-06-12:current:bcf51a4db7e85c094bd39c5e4d320925b1299982fe34778adfcef177a9ce9ddf** — PubMed; class A. [Open source](https://pubmed.ncbi.nlm.nih.gov/1851003/) (`pubmed-observation-pmid-1851003-2019-06-12-bcf51a4db7e8`)

## Correction lineage

No correction is recorded for this version.

## Reporting boundaries and unknowns

PeptideScanner reproduces bounded bibliographic metadata attributed to PubMed. It does not summarize or endorse the article's findings.

- Sources outside the selected public PeptideScanner bundle may exist and are not represented by this object.
- This object does not determine whether cited records agree, corroborate one another, or establish scientific truth.
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## Suggested citation

PeptideScanner. "N-terminal degradation of ACTH(4-10) and its synthetic analog semax by the rat blood enzymes." Version 1, published 2026-08-13. https://peptidescanner.com/evidence/v1/objects/pubmed-pmid-1851003-semax-v1.json SHA-256: 2f3694be65df34a5700e6601ccbd6cc6b40afb12f566d6c526984b57b932ee72.